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CPD Accredited 3rd Annual Congress on Epigenetics & Chromatin (aac) A

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CPD Accredited 3rd Annual Congress on Epigenetics & Chromatin

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Innovation & Discoveries in fields of Epigenetics & Chromatin will lead to be better future

The Epigenetics Congress 2020, Berlin Germany in the month of April 23-24, 2020 will focus on current advances in the research and Technology of Epigenetics & Chromatin with the whole concept of this advanced technology is to process from past, analyse the present and implement for the future the latest innovative evolving theories and technologies to surpass the hurdles and make modish frontiers. Epigenetics & Chromatin Conference will be a multidisciplinary gathering and present of areas such as 3rd Annual Congress on Epigenetics & Chromatin New Technology’s, Education and policies. The forum of Scientists, students and researchers from all corners of the globe, come together to discuss future on science & Technology. Every topic of the meeting will be included with eminent people’s lectures, poster and discussions, join us to design sustainable development processes, innovations by which and how these technologies drive new strategies, advances the business and human health protection. We are glad to invite you on behalf of organizing committee to join us, where you are the decision maker for future.

3rd Annual Congress on Epigenetics & Chromatin Conference purpose is to fill your head with the knowledge you can use: ideas, new trends, and amazing ingenuity. Our focus is on sustainable Development and New Technologies, which we believe are foundation to the success of individual organizations as well as our cities, states, nations, and world. Attendees come to 3rd Annual Congress on Epigenetics & Chromatin to learn from experts in their community and leave.

Conference Highlights

  • Epigenetics
  • Epigenetics Mechanism
  • Epigenetic Diseases
  • Cyto-epigenetic approaches
  • Chromatin & its Dynamics
  • Environmental Epigenetic Factor
  • Genetics & Epigenetic inheritance
  • Microbial Epigenetics
  • Plant Epigenetics
  • Toxic epigenetics
  • Cancer Epigenetics
  • Epigenetics in Clinical Practice
  • Epigenetic Biomarkers in Cancer
  • Chromatin Immunoprecipitation
  • Structure and Function of Chromatin
  • Chromatin Remodelling
  • Chromatin and Epigenetics
  • Immunology in the epigenetics

Sessions / Tracks

Session 1: Epigenetics

Epigenetics is the study of the chemical change of specific genes or gene-associated proteins of an organism. Epigenetic changes can define how the information in genes is expressed and used by cells. other way of looking epigenetics is like this the traditional genetics describes the way the DNA order in our genes are transferred from one generation to the next generation, epigenetics describes transfer on the way the genes are used. To make a computer technology, think of epigenetics as data, information describing and ordering the underlying data

Session 2: Epigenetics Mechanism

Epigenetic mechanisms that helps to regulate gene activity in the CNS to be involved exclusively in developmental processes or in disease states. Recent work states that these mechanisms, mainly for post-translational changes of histones and repulsion changes of DNA, remain labile through the lifespan and are altered by experiences.

Epigenetic mechanisms are useful for controlling gene expression and chromatin designing in mammalian cells, and not surprisingly they play critical roles in both normal cardiac development and heart diseases. Epigenomics is the study of epigenetic features at a genome-wide level.

Session 3: Epigenetic Diseases

Epigenetic changes are responsible for human diseases, including Fragile X syndrome, Angelman’s syndrome, Prader-Willi syndrome, and various cancers. Abbreviations: ATR-X syndrome, alpha-thalassaemia, mental retardation syndrome, X linked; BWS, Beckwith–Wiedemann syndrome; CREB, response-element-binding protein; HAT, histone acetyltransferase; HMT, histone methyltransferase; ICF, immunodeficiency, centromeric region instability and facial anomalies syndrome; UTR, untranslated region.

The role of epigenetics in human diseases has been founded from a half of century ago. In the last decade, especially in complicated disorders such as behaviour plasticity, memory, cancer, autoimmune disease, and addiction as well as neurodegenerative and psychological disorders

Session 4: Cyto-epigenetic approaches

The combination of cytogenetic & epigenetic approaches in chronic lymphocytic leukemia improves prognosis prediction for patients. Cyto genetics is study of chromosomal structure, location and function in cells. Modern cytogenetic approaches are enable to label the chromosomal location of any gene using different colored dots, examine cells from any type of tissue (even tumour cells), identify cells that have lost or gained a specific chromosome & determine whether specific regions of chromosomes have been lost or gained without ever looking at the chromosomes under a microscope.

Cancer cyto genetics


Fluorescent in situ hybridization


Molecular cyto genetics

Session 5: Chromatin & its Dynamics

The dynamics of chromatin has long been of interest to geneticists and cell biologists. For example, the question of whether chromosome rearrangements observed during the pairing of meiotic homologs in maize and Neurospora require special motile machinery, or whether they move by diffusion. The development of fluorescent live cell imaging techniques in recent decades has allowed for chromatin dynamics to be studied in the live cells of a wide range of organisms from yeasts to insects and mammals. When huge chromatin and centromeres studied on the scale of the nucleus, excluding apparent curvilinear chromosome movements that have been attributed to nuclear rotation

Session 6: Environmental Epigenetic Factor

In last century, it was known that DNA by itself does not determine all characteristics of an organism, including humans. The environment, stress one perceives, and nutrition, play a vital part in determining the response of an organism, as much as the DNA itself. Thus, it is known now that both nature (genetic makeup) and nurture (environmental factors) play equally important roles in the responses observed both at the cellular and organism levels.

Thus, human beings are affected by both genetic and epigenetic factors. Some environmentally induced changes in the epigenome are recorded in genomic DNA methylation patterns for up to three generations.

Session 7: Genetics & Epigenetic inheritance

An important set of phenomena, termed epigenetic inheritance, seem to be due to heritable alterations in which the DNA sequence itself is unchanged. Indeed, it is likely that these phenomena constitute another, poorly understood level of gene control. Examples of epigenetic inheritance in which the activity state of a gene depends on its genealogical history are paramutation and parental imprinting.

Epigenetic inheritance is an unconventional finding. It goes averse the idea that inheritance happens only through the DNA code that passes from parent to offspring. It means a parent's experiences, in the form of epigenetic tags, can be passed down to future generations.

Session 8: Microbial Epigenetics

The bacteria can affect the chromatin structure and transcriptional program of host cells by influencing diverse epigenetic factors. Bacterial & Viral infections are involved in the development of human cancers, such as liver, cervical, head and neck, nasopharyngeal and gastric cancers.

DNA methylation affects many biological processes in microbes and may play a role in pathogenicity and virulence. methyltransferase & DNA methylation specificities is possible during long-read sequencing.

Bacteriophage Infection

Bacterial Virulence

Session 9: Plant Epigenetics

The study of epigenetics in plants has a long history, from starting descriptions of non-Mendelian gene behaviors to seminal discoveries of chromatin-modifying proteins and RNAs that mediate gene silencing in most eukaryotes, including humans. Genetic screens in the model plant Arabidopsis have been particularly identifying more than 130 epigenetic regulators . The diversity of epigenetic in plants is remarkable, presumably contributing to the phenotypic plasticity of plant postembryonic development and the ability to survive and reproduce in unpredictable environments.

Session 10: Toxic epigenetics

In the mammalian life cycle evaluates the evidence for environmentally induced epigenetic toxicity in human cohorts and rodent models and highlights the research considerations and implications of this emerging knowledge for public health and regulatory toxicology. Hundreds of studies investigated toxicity, few demonstrated a mechanistic association among specific environmental exposures, epigenetic changes and adverse health outcomes in human epidemiological cohorts and/or rodent models. while, small body of evidence is highly composed of investigative in high-dose range studies, it does set a precedent for the existence of environmentally induced epigenetic toxicity

Session 11: Cancer Epigenetics

Cancer epigenetics is study of somatically changes to molecular processes that effect the flow of information between the DNA of cancer cells and their gene expression patterns. This includes comparative (tumour cell versus normal cell) investigation of nuclear organization, DNA methylation, histone modification and the consequences of genetic mutations in genes encoding epigenetic regulators.

The initiation and advance of cancer, traditionally seen as a genetic disease, is now realized to involve epigenetic abnormalities along with genetic alterations

Session 12: Epigenetics in Clinical Practice

Randomized trials have clearly demonstrated that the hypomethylating agents azacitidine and decitabine are more effective than 'best supportive care'(BSC) in reducing transfusion frequency in 'low-risk' myelodysplasia (MDS) and in prolonging survival compared with BSC or low-dose ara-C in 'high-risk' MDS or acute myeloid leukemia (AML) with 21-30% blasts. They also appear equivalent to conventional induction chemotherapy in AML with >20% blasts and as conditioning regimens before allogeneic transplant (hematopoietic cell transplant, HCT) in MDS.

Session 13: Epigenetic Biomarkers in Cancer

Epigenetic regulation is involved almost all growth processes of mammalian cells from fertilisation, implantation, and differentiation during embryonic advancement to aging and carcinogenesis. Different stages of development are characterized by differences in epigenetic signatures, and variations in epigenetic patterns may also be associated with specific stages of disease. Carcinogenesis is a multistep process; the detection of changes in epigenetic profiles can be exploited to differentiate not only between different types of malignancies but also between different stages of cancer progression. Epigenetic biomarkers hold great promise to become more conclusive diagnostic and prognostic biomarkers for different cancers. DNA methylation is the most studied area of epigenetics; DNA methylation biomarkers will play a prominent role. Epigenetic regulation plays a major role in cancer formation, and analysis of epigenetic biomarkers has great potential to become clinically relevant.

Session 14: Chromatin Immunoprecipitation

Chromatin immunoprecipitation (ChIP) is a technique it determines a protein of interest interacts with a specific DNA sequence. This technique is often used to study the repertoire of sites on DNA that are bound by particular transcription factors or by histone proteins, and to look at the precise genomic locations of various histone modifications (including acetylation, phosphorylation, or methylation). ChIP can be used to analyse the presence of protein-DNA interaction at steady state, Protein and associated chromatin are temporarily cross-linked in live cells or tissues (using formaldehyde or UV) and sheared using enzymatic digestion

Session 15: Structure and Function of Chromatin

The function of chromatin is to systematic package DNA into a small volume to fit into the nucleus of a cell and protect the DNA structure and sequence. Packaging DNA into chromatin allows for mitosis and meiosis, stops chromosome breakage and controls gene expression and DNA replication.

DNA wraps around the histone proteins to form nucleosomes, it turn couple to become the chromatin fiber.

1) Unpackaged DNA.

2) DNA wrapped around histone octamers to form nucleosomes.

3) Nucleosomes compacted into a chromatin fiber.

Session 16: Chromatin Remodelling

The variety of chromatin remodeling complexes that exist allow for a number of different mechanisms to remodel chromatin. Remodelers can slide nucleosomes, eject histone octamers, and replace dimers . These work require breakage of all 14 histone-DNA contacts and require approximately 12–14 kcal mol?1 (Gottesfeld and Luger 2001). All chromatin remodeler ATPases are members of the SF2 family of DEAD/H-box helicase/tranlocases and are in fact ATP-dependent DNA translocases (Cairns 2007; Saha et al. 2002; Whitehouse et al. 2003). Remodelers share common properties but are dedicated for specific tasks.

Session 17: Chromatin and Epigenetics

The epigenetic regulation of chromatin structure and composition has been studied molecularly in specific DNA-dependent processes. epigenetics play important global roles in moulding and maintaining cell identity, and in patterning the body plan during normal development. the alterations in epigenetic regulation are involved in many diseases, including cancer. The advances in our understanding of the impact of epigenetics in development and disease were discussed at a recent Keystone symposium. The importance of molecular machines that act on chromatin to regulate gene expression has fuelled a great interest in this field. it clear that epigenetics does not only affect the expression of individual genes.

Session 18: Immunology in the epigenetics

Recently, it has been well documented that epigenetic mechanisms like DNA methylation and histone changes control the expression of immune system-related genes, changing the development of the innate and adaptive immune responses. An in-depth knowledge of these epigenetic mechanisms could balance the immune response after transplantation and to develop new therapeutic strategies . The detection of epigenetic marks in main immune genes could be useful as biomarkers of rejection and progression among transplanted patients.


Coming Soon!

Organizing Committee

AC Matin

Stanford University

Santa clara, USA

Wassil Nowicky

Director Nowicky Pharma & President -Ukrainian Anti-Cancer Institute

Ukrainian Anti-Cancer Institute

Vienna, Austria

Dr. Joel I. Osorio

CEO & Co-Founder

RegenerAge Clinic

Mexico, Mexico

Gerald C. Hsu

EclaireMD Foundation

Indiana, USA

Henry M. Sobell


University of Rochester, USA

Los Angeles, USA

Zeev Blumenfeld, MD

Associate Professor, Reproductive endocrinology/Ob.Gyn.

MEUHEDETH Med Services

Israel, Israel

Darshit A Thaker


Queensland, Australia

Kadri Altundag

MKA Breast Cancer Clinic

Istanbul, Turkey

Dr. Masahiro Onuma


Trisguide ltd

Tokyo, Japan

Vijay Sharma

Oxford Healthcare Innovation ( OX HI )

Jaipur, India

Prof. Dr. C.P Abdolrasoul Aleezaadeh

Payame Noor University(PNU) and, University of Applied Science and Technology (U.A.S.T)

Tehran, Iran

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